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E cadherin breast cancer

The journal is indexed in many databases, though not in PubMed. Here, we examined the correlation between Twist, E-cadherin, and estrogen receptor ER in promoting invasion and metastasis in breast cancer cells and tissues. Figure 4: The protein expression of Twist, E-cadherin, and estrogen receptor in breast cancer tissues and adjacent tissues. Tang D. Genetic polymorphisms are the change of the common DNA sequence in the general population. Matthews J, Gustafsson JA.
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CDH1 (gene)

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JCDR - E-Cadherin, Breast carcinoma

Cancer Res ; Cadherin cell adhesion receptors as a morphogenetic regulator. Scores higher than six were considered high quality. It is well said that "happy beginning is half done" and it fits perfectly with JCDR. Table 1 CDH1 gene exon 1—16 mutations in breast cancer. Volume 3 Issue 4 Dec , pp. There is fast growing authorship and readership with JCDR as this can be judged by the number of articles published in it i e; in Feb of its first issue, it contained 5 articles only, and now in its recent volume published in April , it contained 67 manuscripts.
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Open Life Sciences

Over a span of over four years, we I and my colleagues have published around 25 articles in the journal. We further performed sensitivity analysis to gauge the stability of our results with respect to OS, DFS, and clinicopathological characteristics. In this editorial, I plan to briefly discuss my experiences of publishing with JCDR and the strengths of the journal and to finally address the areas for improvement.
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Perry C. The prepublication and the manuscript tracking system are also helpful for the authors. To publish a journal manuscripts are needed from authors. It is well-known that the functional loss of E-cadherin has been viewed as the most important hallmark of epithelial—mesenchymal transition EMT , which induces tumor cell dissemination and subsequently increases cell migration and invasion [ 45 , 46 ]. Watch next beautiful korean porn
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